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Neuromuscular

Cervical Disc Herniation Treatment in Tel Aviv

Cervical disc herniation affects 40-60 per 100,000 annually. Radiculopathy (nerve root compression) symptoms resolve in 75-90% within 12 months using conservative management. C6 and C7 nerve roots account for 85% of cases.

75-90% conservative success at 12 months
Equal outcomes physio vs surgery at 1 year
C7 60% · C6 25% of radiculopathy
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Cervical Levels & Nerve Root Patterns

Disc herniation location determines clinical presentation. C6 and C7 nerve roots account for 85% of symptomatic cases (Radhakrishnan et al., Brain 1994). The foraminal diameter narrows by 20-30% during extension and ipsilateral lateral flexion — which is why these movements are typically provocative.

Level
Nerve Root
Pain Pattern
Weakness
Reflex Loss
C3-C4
C4
Neck + shoulder girdle referral
Diaphragm (rare)
None
C4-C5
C5
Lateral arm, deltoid
Shoulder abduction, deltoid
Biceps
C5-C6
C6
Lateral forearm, thumb + index
Wrist extensors, biceps
Brachioradialis★COMMON
C6-C7
C7
Middle finger, posterior arm
Triceps, wrist flexors
Triceps★MOST COMMON
C7-T1
C8
Ring + little finger, medial forearm
Finger flexors, intrinsics
None

Red Flags: Radiculopathy vs Myelopathy

Myelopathy (spinal cord compression) is a surgical emergency. Identify and refer immediately if any of these signs are present.

Myelopathy Red Flags — Seek Urgent Imaging

  • Both arms weak or numb (not dermatomal)
  • Gait disorder, clumsiness, balance loss
  • Hand weakness (button-pushing difficulty, fine motor loss)
  • Bowel or bladder dysfunction
  • Hoffman sign positive · Babinski sign positive · clonus · hyperreflexia

The Science of Cervical Disc Herniation

A cervical disc consists of two components: the nucleus pulposus — a gel-like inner core rich in proteoglycans and water (80% water in a healthy disc) — and the annulus fibrosus, a series of 12-20 concentric fibrocartilaginous rings surrounding it. The nucleus acts as a hydraulic shock absorber, distributing compressive forces evenly across the disc. With age, repeated loading, or acute trauma, the annulus develops fissures, allowing the nucleus to migrate outward (prolapse) or fully breach the annular wall (extrusion).

A herniation does not necessarily mean nerve compression: the degree of inflammatory mediators released (PLA2, IL-1β, TNF-α) around the herniated material is a stronger predictor of radicular symptoms than size alone. This is why some large herniations on MRI are asymptomatic, while smaller ones can produce severe pain.

Spurling Test & Clinical Assessment

The Spurling test (specificity 93%, sensitivity 30-50%) reproduces radicular pain and confirms nerve root irritation. Manoeuvre: ipsilateral rotation + extension + gentle axial compression. A positive test = arm pain in dermatomal pattern.

  • Distraction test: relieves arm pain with axial unloading (sensitivity 40-50%, specificity 80-100%)
  • ULTT (Upper Limb Tension Test): assesses neural mobility — pain reproduction = positive
  • Neurological screen: reflexes (biceps C5-6, brachioradialis C6, triceps C7), myotomes, dermatomes
  • Imaging (MRI): indicated for progressive deficit, myelopathy signs, or failure of 6-12 weeks conservative care

Clinical Pearl: Radiculopathy pain + dermatomal pattern + positive Spurling = nerve root compression. No imaging needed to start conservative treatment. 75-90% resolve in 12 months without surgery.

Treatment Hierarchy: Conservative First

Sampath 2000 (JBJS, n=1550): 75-90% of cervical radiculopathy resolves conservatively at 12 months. Peul 2007 (NEJM): surgery accelerates relief by 4-6 weeks but outcomes are equal at 1 year. Conservative care is the recommended first-line approach.

  • Phase 1 (Weeks 1-2): gentle cervical traction (intermittent, 5-10 min), isometric neck stabilisation, scapular stabilisation, pain modulation
  • Phase 2 (Weeks 3-4): neural mobilisation (ULTT progression), cervical ROM exercises, postural correction, McKenzie directional preference
  • Phase 3 (Weeks 5-8): progressive strengthening (isometric → isotonic), deep neck flexor retraining (Jull protocol), functional training
  • Phase 4 (Weeks 9-12): sport-specific drills, cervical stability load progression, return-to-activity, recurrence prevention

Neural Mobilisation Techniques

Neural mobilisation addresses mechanosensitivity of the brachial plexus and peripheral nerves. Two approaches:

Slider techniques move the nerve bed in opposite directions simultaneously (e.g., elbow flexion while depressing the shoulder), creating a sliding motion through surrounding tissues without increasing tension. Used in the acute/subacute phase when the nerve is highly irritable. Tensioner techniques increase tension throughout the entire neural pathway and are reserved for chronic or less irritable presentations. Nee & Butler (Man Ther 2013) found neural mobilisation reduces arm pain by a mean of 2.3/10 VAS with moderate effect size.

  • ULTT1 (median nerve): shoulder abduction + external rotation → elbow extension → forearm supination → wrist extension
  • ULTT2 (radial nerve): similar but with wrist flexion and thumb adduction
  • ULTT3 (ulnar nerve): elbow flexion with wrist extension and ulnar deviation

Cervical Traction Protocols

Mechanical traction enlarges the intervertebral foramen by 1-2mm and decompresses the nerve root. Indicated in radiculopathy with positive Distraction test. A Cochrane review (Graham et al., 2008) found moderate evidence for short-term pain relief when combined with manual therapy and exercise.

  • Intermittent traction: 7-12 kg, 25-35° flexion, 15-30 sec hold, 2-3× per session
  • Sustained traction: 10-15 lbs, 10-20 min holds, once weekly after acute phase
  • Contraindicated if myelopathy, vertebral instability, or rheumatoid arthritis affecting C-spine
  • Combine with postural correction, deep neck flexor training, and ROM exercises

Radiculopathy or Myelopathy?

Evidence-based assessment determines conservative vs surgical pathway. 75-90% of radiculopathy resolves without surgery — accurate diagnosis is the first step.

Return to Work and Activity

Continued activity (with ergonomic modification) leads to better outcomes than sick leave for cervical disc herniation. Key workplace adjustments include: monitor height at eye level to reduce cervical extension loading, document holders adjacent to the screen to avoid sustained neck rotation, and positional changes every 30-45 minutes. Full return to pre-injury activity is expected in 85-90% of patients treated conservatively at 3-6 months (Sampath et al., JBJS 2000).

Frequently Asked Questions

Radiculopathy = single nerve root compression (unilateral arm pain, dermatomal pattern). Myelopathy = spinal cord compression (bilateral arm symptoms, gait disorder, hand weakness, hyperreflexia, Hoffman/Babinski signs). Myelopathy is a red flag requiring urgent imaging and possible surgery.
Yes. 75-90% of cervical radiculopathy resolves with conservative treatment at 12 months (Sampath 2000, n=1550). Surgery accelerates relief by 4-6 weeks but outcomes are equal at 1 year (Peul NEJM 2007). Conservative care is recommended first-line.
Clinical test for cervical radiculopathy: ipsilateral rotation + extension + axial compression. Specificity 93%. Positive test = radicular arm pain reproducing the nerve root irritation pattern in dermatomal distribution.
Acute radiculopathy: 4-6 weeks with 2-3× per week sessions. Chronic: 8-12 weeks. Most patients achieve significant pain reduction by week 4, with full resolution by 12 weeks if compliant with home exercises.
Surgery indicated if: failed conservative treatment (>12 weeks), progressive myelopathy (bilateral symptoms, gait loss), severe functional loss preventing work/sleep, or MRI evidence of cord compression with progressive symptoms. Otherwise, 75-90% resolve conservatively.

Cervical Disc Herniation Holding You Back?

Evidence-based conservative treatment resolves 75-90% of radiculopathy in 12 months. Specialised neural mobilisation, traction, and progressive strengthening protocols.

Clinical information · Recovery TLV

WHAT IS IT — Cervical disc herniation is protrusion or extrusion of nucleus pulposus through annular fissures, most commonly at C5-C6 and C6-C7 (85% of cases). Herniated material releases inflammatory mediators (PLA2, IL-1β, TNF-α) causing chemical radiculitis independent of mechanical compression. Foramen narrows 20-30% in extension and ipsilateral lateral flexion. Spurling test: sensitivity 30-50%, specificity 93%. ULTT median nerve bias: high sensitivity for median nerve tension. Myelopathy (cord compression) = red flag for urgent surgical referral.

WHO IT AFFECTS — Cervical radiculopathy incidence: 83/100,000/year (Radhakrishnan et al., Brain 1994). C7 most common (60%), C6 (25%). Spontaneous remission at 12 months: 75-90% (Sampath et al., JBJS 2000). Surgery equivalent to physio at 12 months (Peul et al., NEJM 2007) but faster early relief. In Tel Aviv: tech workers (sustained flexion/rotation), cyclists, swimmers with repetitive neck rotation.

HOW WE TREAT IT — Recovery TLV MDT-based approach: directional preference testing (centralisation in 55-70% of radiculopathy — positive prognostic sign). Neural mobilisation (slider → tensioner progression). Cervical traction (15-20 min, 7-12kg at 25-35°). Deep neck flexor retraining. Activity modification (avoid sustained flexion, screen height). Referral criteria: myelopathy signs (Hoffman, clonus, bilateral UL symptoms), progressive motor deficit, failure of 6-12 weeks conservative care.

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